Arteriosclerosis, Thrombosis, and Vascular Biology -- Table of Contents (27 [8])

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Volume 27, Issue 8; August 1, 2007

Editorials
Brief Reviews
Vascular Biology
Atherosclerosis and Lipoproteins
Thrombosis
Letters to the Editor

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	Editorials
	Hormone Replacement Therapy and Atherosclerosis in Postmenopausal Women: Does Aging Limit Therapeutic Benefits? Matthias Barton, Matthias R. Meyer, and Elvira Haas Arterioscler Thromb Vasc Biol. 2007;27:1669-1672, doi:10.1161/ATVBAHA.106.130260 Full Text \| PDF
	Combating Nitrate Tolerance: A Novel Endogenous Mechanism David D. Gutterman Arterioscler Thromb Vasc Biol. 2007;27:1673-1676, doi:10.1161/ATVBAHA.107.148023 Full Text \| PDF
	A No-No for NonO and JNK in Extracellular Matrix Homeostasis and Vascular Stability David Y. Hui Arterioscler Thromb Vasc Biol. 2007;27:1677-1678, doi:10.1161/ATVBAHA.107.146894 Full Text \| PDF
	Emerging Role of IGF-1R in Stretch-Induced Neointimal Hyperplasia in Venous Grafts Hannah Song, Amy L. Mowbray, Michelle C. Sykes, and Hanjoong Jo Arterioscler Thromb Vasc Biol. 2007;27:1679-1681, doi:10.1161/ATVBAHA.107.148189 Full Text \| PDF
	Summary of the American Heart Association’s Scientific Statement on the Relevance of Genetics and Genomics for Prevention and Treatment of Cardiovascular Disease Donna K. Arnett for the Writing Group Arterioscler Thromb Vasc Biol. 2007;27:1682-1686, doi:10.1161/ATVBAHA.107.147207 Full Text \| PDF
	Brief Reviews
	Role of the Extrinsic Pathway of Blood Coagulation in Hemostasis and Thrombosis Nigel Mackman, Rachel E. Tilley, and Nigel S. Key Arterioscler Thromb Vasc Biol. 2007;27:1687-1693; published online before print June 7 2007, doi:10.1161/ATVBAHA.107.141911 Abstract \| Full Text \| PDF The extrinsic pathway of blood coagulation is required for thrombosis. However, under pathologic conditions, TF expression within the vasculature leads to thrombosis. Development of safe and effective hemostatic agents and antithrombotic drugs requires a better understanding of the role of TF and FVIIa in hemostasis and thrombosis.
	Peripheral Circadian Clocks in the Vasculature Dermot F. Reilly, Elizabeth J. Westgate, and Garret A. FitzGerald Arterioscler Thromb Vasc Biol. 2007;27:1694-1705; published online before print May 31 2007, doi:10.1161/ATVBAHA.107.144923 Abstract \| Full Text \| PDF Endogenous circadian clocks are present in the cardiovascular system. At the molecular level output from these oscillators manifests as circadian expression of transcripts for many genes relevant to cardiovascular disease. In this review we discuss the potential contribution of these molecular oscillators to the observed diurnal variation in cardiovascular disease.
	Mouse Models for Atherosclerosis and Pharmaceutical Modifiers Susanne Zadelaar, Robert Kleemann, Lars Verschuren, Jitske de Vries-Van der Weij, José van der Hoorn, Hans M. Princen, and Teake Kooistra Arterioscler Thromb Vasc Biol. 2007;27:1706-1721; published online before print May 31 2007, doi:10.1161/ATVBAHA.107.142570 Abstract \| Full Text \| PDF This review highlights some frequently used mouse models for atherosclerosis research and compares their responses to selective antiatherosclerotic manipulation.
	Vascular Remodeling: Hemodynamic and Biochemical Mechanisms Underlying Glagov’s Phenomenon Vyacheslav A. Korshunov, Stephen M. Schwartz, and Bradford C. Berk Arterioscler Thromb Vasc Biol. 2007;27:1722-1728; published online before print May 31 2007, doi:10.1161/ATVBAHA.106.129254 Abstract \| Full Text \| PDF This review focuses attention on the need to identify specific molecular pathways that explain the relationship of physical parameters and arterial remodeling. Recent studies have begun to define the transduction pathways in the artery wall that determine their ability to respond to the physical forces exerted by blood flow.
	Vascular Biology
	Heme Oxygenase-1: A Novel Key Player in the Development of Tolerance in Response to Organic Nitrates Philip Wenzel, Matthias Oelze, Meike Coldewey, Marcus Hortmann, Andreas Seeling, Ulrich Hink, Hanke Mollnau, Dirk Stalleicken, Henry Weiner, Jochen Lehmann, Huige Li, Ulrich Förstermann, Thomas Münzel, and Andreas Daiber Arterioscler Thromb Vasc Biol. 2007;27:1729-1735; published online before print May 31 2007, doi:10.1161/ATVBAHA.107.143909 Abstract \| Full Text \| PDF \| Data Supplement Tolerance to nitroglycerin is likely attributable to an increased production of reactive oxygen species and an inhibition of its bioactivating enzyme. Pentaerythrityl tetranitrate (PETN) therapy is devoid of tolerance, potentially because of HO-1 induction. Inhibition of HO-1 expression induced "tolerance" to PETN, whereas HO-1 induction prevented tolerance in nitroglycerin-treated rats.
	Nox1 Mediates Basic Fibroblast Growth Factor-Induced Migration of Vascular Smooth Muscle Cells Katrin Schröder, Ina Helmcke, Katalin Palfi, Karl-Heinz Krause, Rudi Busse, and Ralf P. Brandes Arterioscler Thromb Vasc Biol. 2007;27:1736-1743; published online before print May 31 2007, doi:10.1161/ATVBAHA.107.142117 Abstract \| Full Text \| PDF The role of reactive oxygen species (ROS) generated by NADPH oxidase for basic fibroblast growth factor (bFGF)-induced smooth muscle cell migration was studied. bFGF-induced migration was dependent on ROS generated from the NADPH oxidase Nox1 but not Nox4 via a pathway involving JNK and paxillin.
	Mechanical Stretch Simulates Proliferation of Venous Smooth Muscle Cells Through Activation of the Insulin-Like Growth Factor-1 Receptor Jizhong Cheng and Jie Du Arterioscler Thromb Vasc Biol. 2007;27:1744-1751; published online before print May 31 2007, doi:10.1161/ATVBAHA.107.147371 Abstract \| Full Text \| PDF In vein VSMCs, mechanical stretch induces proliferation and activation of IGF-1R. Conditional null deletion of IGF-1R in mice reduced the formation of neointima in the vein graft. Our results have uncovered a signaling pathway that leads to neointima formation in vein graft.
	Interferon- ${gamma}$ Suppresses Cyclooxygenase-2 Promoter Activity by Inhibiting C-Jun and C/EBPß Binding Wu-Guo Deng, Alberto J. Montero, and Kenneth K. Wu Arterioscler Thromb Vasc Biol. 2007;27:1752-1759; published online before print June 7 2007, doi:10.1161/ATVBAHA.107.144352 Abstract \| Full Text \| PDF \| Data Supplement INF-{gamma} suppressed proinflammatory mediator-induced transcriptional activation in human cells by selective inhibition of C-Jun and C/EBPß binding to their respective response elements on COX-2 promoter. We propose that IFN{gamma} may represent an important factor for controlling COX-2-mediated vascular inflammatory diseases including the development of atheromatous plaque.
	TNF- ${alpha}$ Suppresses Prolyl-4-Hydroxylase ${alpha}$ 1 Expression via the ASK1–JNK–NonO Pathway Cheng Zhang, Ming-Xiang Zhang, Ying H. Shen, Jared K. Burks, Yun Zhang, Jian Wang, Scott A. LeMaire, Koichi Yoshimura, Hiroki Aoki, Joseph S. Coselli, and Xing Li Wang Arterioscler Thromb Vasc Biol. 2007;27:1760-1767; published online before print May 3 2007, doi:10.1161/ATVBAHA.107.144881 Abstract \| Full Text \| PDF We showed that TNF-A dose-dependently downregulated the P4H{alpha}(I)--the rate-limiting subunit for the P4H enzyme essential for procollagen hydroxylation, secretion, and deposition. Using a progressive deletion cloning approach, we characterized the TNF-A-responsive element in the P4H{alpha}(I) promoter and identified the NonO protein may be responsible for the suppressing effect.
	Increased Inhibition of Inward Rectifier K⁺ Channels by Angiotensin II in Small-Diameter Coronary Artery of Isoproterenol-Induced Hypertrophied Model Won Sun Park, Jae-Hong Ko, Nari Kim, Youn Kyoung Son, Sung Hyun Kang, Mohamad Warda, In Duk Jung, Yeong-Min Park, and Jin Han Arterioscler Thromb Vasc Biol. 2007;27:1768-1775; published online before print May 24 2007, doi:10.1161/ATVBAHA.107.143339 Abstract \| Full Text \| PDF \| Data Supplement Kir current amplitude was definitely lower in the coronary arterial smooth muscle cells of Iso-induced hypertrophied model than in the control. Ang II inhibits Kir current, which is more prominent in Iso-induced hypertrophied model owing to increases in the expression of AT1 receptor and the activation of PKC{alpha}. The results help for better understanding the responsiveness of small-diameter arteries during hypertrophy.
	Increased Uridine Adenosine Tetraphosphate Concentrations in Plasma of Juvenile Hypertensives Vera Jankowski, Andreas-Alexander Meyer, Peter Schlattmann, Yu Gui, Xi-long Zheng, Irini Stamcou, Kristina Radtke, Thi Nguyet Anh Tran, Markus van der Giet, Markus Tölle, Walter Zidek, and Joachim Jankowski Arterioscler Thromb Vasc Biol. 2007;27:1776-1781; published online before print June 14 2007, doi:10.1161/ATVBAHA.107.143958 Abstract \| Full Text \| PDF This study describes the association of circulating levels of Up4A with juvenile hypertension. Up4A concentration correlated with blood pressure, intima media wall thickness, and left ventricular mass.
	Atherosclerosis and Lipoproteins
	Age Moderates the Short-Term Effects of Transdermal 17ß-Estradiol on Endothelium-Dependent Vascular Function in Postmenopausal Women Andrew Sherwood, Julie K. Bower, Judith McFetridge-Durdle, James A. Blumenthal, L. Kristin Newby, and Alan L. Hinderliter Arterioscler Thromb Vasc Biol. 2007;27:1782-1787; published online before print May 31 2007, doi:10.1161/ATVBAHA.107.145383 Abstract \| Full Text \| PDF We sought to evaluate whether age and coronary heart disease moderate the effects of 17ß-estradiol on vascular endothelial function. For younger postmenopausal women, short-term estradiol transdermal patch exposure was associated with improved flow mediated dilation. Benefits of estrogen supplementation on vascular endothelial function may be dependent on postmenopausal age.
	Oxidized Phospholipids, Lipoprotein(a), Lipoprotein-Associated Phospholipase A2 Activity, and 10-Year Cardiovascular Outcomes: Prospective Results From the Bruneck Study Stefan Kiechl, Johann Willeit, Manuel Mayr, Brigitte Viehweider, Martin Oberhollenzer, Florian Kronenberg, Christian J. Wiedermann, Sabine Oberthaler, Qingbo Xu, Joseph L. Witztum, and Sotirios Tsimikas Arterioscler Thromb Vasc Biol. 2007;27:1788-1795; published online before print May 31 2007, doi:10.1161/ATVBAHA.107.145805 Abstract \| Full Text \| PDF Oxidized phospholipids (OxPL) detected by monoclonal antibody E06 (OxPL/apoB), lipoprotein(a), and Lp-PLA2 activity were measured in the Bruneck Study, where 82 of 765 subjects developed cardiovascular events over 10 years. OxPL/apoB and Lp(a) predicted 10-year cardiovascular events independently of traditional risk factors, hsCRP, and the Framingham risk score. Increasing Lp-PLA2 activity further amplified the risk mediated by OxPL/apoB and Lp(a).
	Serum Adipocyte Fatty Acid-Binding Protein Levels Were Independently Associated With Carotid Atherosclerosis D.C.Y. Yeung, A. Xu, C.W.S. Cheung, N.M.S. Wat, M.H. Yau, C.H.Y. Fong, M.T. Chau, and K.S.L. Lam Arterioscler Thromb Vasc Biol. 2007;27:1796-1802; published online before print May 17 2007, doi:10.1161/ATVBAHA.107.146274 Abstract \| Full Text \| PDF This study investigated the relationship between serum adipocyte fatty acid-binding protein (A-FABP) levels and carotid intima-media thickness (IMT) in 479 Chinese subjects. It was found that serum A-FABP levels correlated positively with carotid IMT in both sexes. Multiple regression analysis showed that serum A-FABP levels were independently associated with carotid IMT in women.
	A Systematic Review and Meta-Analysis of Statin Therapy in Children With Familial Hypercholesterolemia H.J. Avis, M.N. Vissers, E.A. Stein, F.A. Wijburg, M.D. Trip, J.J.P. Kastelein, and B.A. Hutten Arterioscler Thromb Vasc Biol. 2007;27:1803-1810; published online before print June 14 2007, doi:10.1161/ATVBAHA.107.145151 Abstract \| Full Text \| PDF This meta-analysis supports the notion that statin therapy is efficacious and safe in children with heterozygous familial hypercholesterolemia. Statin-treated children had a less atherogenic lipid profile and did not experience more adverse events, impaired growth and sexual development, or worsened laboratory safety profile when compared with placebo.
	Weight-Related Increases in Hypertension, Hypercholesterolemia, and Diabetes Risk in Normal Weight Male and Female Runners Paul T. Williams, Kathryn Hoffman, and Isabelle La Arterioscler Thromb Vasc Biol. 2007;27:1811-1819; published online before print May 17 2007, doi:10.1161/ATVBAHA.107.141853 Abstract \| Full Text \| PDF Even among normal-weight physically-active individuals (BMI <25 kg/m2), increasing BMI and waist circumference significantly increased the odds for hypertension (P<0.0001), hypercholesterolemia (P<0.0001), and diabetes (P<0.02) in men, and hypertension in women (P=0.05). The odds for being diagnosed with high cholesterol also increased significantly with increasing BMI in women <25 kg/m2.
	Relationship Between a Systemic Inflammatory Marker, Plaque Inflammation, and Plaque Characteristics Determined by Intravascular Optical Coherence Tomography O. Christopher Raffel, Guillermo J. Tearney, Denise DeJoseph Gauthier, Elkan F. Halpern, Brett E. Bouma, and Ik-Kyung Jang Arterioscler Thromb Vasc Biol. 2007;27:1820-1827; published online before print May 31 2007, doi:10.1161/ATVBAHA.107.145987 Abstract \| Full Text \| PDF We compared the relationships between the peripheral white cell count, local plaque fibrous cap macrophage density, and the morphological features and presence of TCFA by using optical coherence tomography. Fibrous cap macrophage density correlated significantly with the peripheral WBC count, and both parameters independently and particularly in combination predict the presence of TCFA.
	Minimal Lipidation of Pre-ß HDL by ABCA1 Results in Reduced Ability to Interact with ABCA1 Anny Mulya, Ji-Young Lee, Abraham K. Gebre, Michael J. Thomas, Perry L. Colvin, and John S. Parks Arterioscler Thromb Vasc Biol. 2007;27:1828-1836; published online before print May 17 2007, doi:10.1161/ATVBAHA.107.142455 Abstract \| Full Text \| PDF \| Data Supplement Incubation of apoA-I with ABCA1-expressing cells generated heterogeneous-sized nascent pre-ß HDLs, which appear to form simultaneously. These particles are poor substrates for ABCA1, suggesting that the initial interaction of apoA-I with ABCA1 results in a constrained conformation of apoA-I and that further maturation of these nascent HDL occurs via non-ABCA1-mediated pathways.
	In Vivo Reverse Cholesterol Transport From Macrophages Lacking ABCA1 Expression Is Impaired Ming-Dong Wang, Vivian Franklin, and Yves L. Marcel Arterioscler Thromb Vasc Biol. 2007;27:1837-1842; published online before print May 31 2007, doi:10.1161/ATVBAHA.107.146068 Abstract \| Full Text \| PDF ATP-binding cassette transporter A1 (ABCA1) is a key mediator of cholesterol efflux to apoA-I in cholesterol loaded macrophages, a first step of reverse cholesterol transport (RCT) in vivo. Here we demonstrate that abca1 inactivation in macrophages significantly impairs in vivo RCT.
	Preferential Sphingosine-1-Phosphate Enrichment and Sphingomyelin Depletion Are Key Features of Small Dense HDL3 Particles: Relevance to Antiapoptotic and Antioxidative Activities Anatol Kontush, Patrice Therond, Amal Zerrad, Martine Couturier, Anne Négre-Salvayre, Juliana A. de Souza, Sandrine Chantepie, and M. John Chapman Arterioscler Thromb Vasc Biol. 2007;27:1843-1849; published online before print June 14 2007, doi:10.1161/ATVBAHA.107.145672 Abstract \| Full Text \| PDF \| Data Supplement Preferential sphingosine-1-phosphate enrichment, but sphingomyelin depletion, distinguish normolipidemic small, dense HDL3 from large HDL2. Elevated sphingosine-1-phosphate/sphingomyelin ratio constitutes a hallmark of the lipid moiety of small dense HDL3 particles and is implicated in their antiatherogenic activity.
	Thrombosis
	Effects of Unfractionated Heparin and Glycoprotein IIb/IIIa Antagonists Versus Bivalirdin on Myeloperoxidase Release From Neutrophils Guohong Li, Alison C. Keenan, Justin C. Young, Margaret J. Hall, Zehra Pamuklar, E. Magnus Ohman, Steven R. Steinhubl, and Susan S. Smyth Arterioscler Thromb Vasc Biol. 2007;27:1850-1856; published online before print May 24 2007, doi:10.1161/ATVBAHA.107.144576 Abstract \| Full Text \| PDF \| Data Supplement Unfractionated heparin+eptifibatide was associated with higher myeloperoxidase levels after percutaneous coronary intervention. Heparin, but not bivalirudin, fondaparinux, or eptifibatide, increased MPO release from and binding to isolated neutrophils. These results indicate that adjuvant therapy during PCI may have undesired effects on neutrophil activation, MPO release, and systemic inflammation.
	A Novel Class of Antioxidants Inhibit LPS Induction of Tissue Factor by Selective Inhibition of the Activation of ASK1 and MAP Kinases James P. Luyendyk, J. Daniel Piper, Michael Tencati, K. Veera Reddy, Todd Holscher, Rong Zhang, Jayraz Luchoomun, Xilin Chen, Wang Min, Charles Kunsch, and Nigel Mackman Arterioscler Thromb Vasc Biol. 2007;27:1857-1863; published online before print June 7 2007, doi:10.1161/ATVBAHA.107.143552 Abstract \| Full Text \| PDF \| Data Supplement We showed that novel compounds with antioxidant and antiinflammatory properties inhibit LPS activation of the redox-sensitive kinase, ASK-1, MAPKs, and the transcription factors AP-1 and Egr-1 without affecting nuclear translocation of NF-{kappa}B. This results in a reduction in TF gene expression in monocytic and endothelial cells.
	Letters to the Editor
	Does Cuff Location for FMD Matter in Smokers? Joshua A. Beckman Arterioscler Thromb Vasc Biol. 2007;27:e140, doi:10.1161/ATVBAHA.107.142828 Full Text \| PDF
	Does Cuff Location for FMD Matter in Smokers? Sashi Guthikonda, Christine A. Sinkey, and William G. Haynes Arterioscler Thromb Vasc Biol. 2007;27:e141, doi:10.1161/ATVBAHA.107.148197 Full Text \| PDF
	Reconstituted HDL Increases Circulating Endothelial Progenitor Cells in Patients With Type 2 Diabetes O. van Oostrom, M. Nieuwdorp, P.E. Westerweel, I.E. Hoefer, R. Basser, E.S.G. Stroes, and M.C. Verhaar Arterioscler Thromb Vasc Biol. 2007;27:1864-1865, doi:10.1161/ATVBAHA.107.143875 Full Text \| PDF
	Prevalence of Cholesteryl Ester Storage Disease Sandro Muntoni, Heiko Wiebusch, Marianne Jansen-Rust, Stephan Rust, Udo Seedorf, Helmut Schulte, Klaus Berger, Harald Funke, and Gerd Assmann Arterioscler Thromb Vasc Biol. 2007;27:1866-1868, doi:10.1161/ATVBAHA.107.146639 Full Text \| PDF

Spotlight

TOC Spotlight File

Volume 27, Issue 8; August 1, 2007

Editorials

Hormone Replacement Therapy and Atherosclerosis in Postmenopausal Women: Does Aging Limit Therapeutic Benefits?

Combating Nitrate Tolerance: A Novel Endogenous Mechanism

A No-No for NonO and JNK in Extracellular Matrix Homeostasis and Vascular Stability

Emerging Role of IGF-1R in Stretch-Induced Neointimal Hyperplasia in Venous Grafts

Summary of the American Heart Association’s Scientific Statement on the Relevance of Genetics and Genomics for Prevention and Treatment of Cardiovascular Disease

Brief Reviews

Role of the Extrinsic Pathway of Blood Coagulation in Hemostasis and Thrombosis

Peripheral Circadian Clocks in the Vasculature

Mouse Models for Atherosclerosis and Pharmaceutical Modifiers

Vascular Remodeling: Hemodynamic and Biochemical Mechanisms Underlying Glagov’s Phenomenon

Vascular Biology

Heme Oxygenase-1: A Novel Key Player in the Development of Tolerance in Response to Organic Nitrates

Nox1 Mediates Basic Fibroblast Growth Factor-Induced Migration of Vascular Smooth Muscle Cells

Mechanical Stretch Simulates Proliferation of Venous Smooth Muscle Cells Through Activation of the Insulin-Like Growth Factor-1 Receptor

Interferon- Suppresses Cyclooxygenase-2 Promoter Activity by Inhibiting C-Jun and C/EBPß Binding

TNF- Suppresses Prolyl-4-Hydroxylase 1 Expression via the ASK1–JNK–NonO Pathway

Increased Inhibition of Inward Rectifier K+ Channels by Angiotensin II in Small-Diameter Coronary Artery of Isoproterenol-Induced Hypertrophied Model

Increased Uridine Adenosine Tetraphosphate Concentrations in Plasma of Juvenile Hypertensives

Atherosclerosis and Lipoproteins

Age Moderates the Short-Term Effects of Transdermal 17ß-Estradiol on Endothelium-Dependent Vascular Function in Postmenopausal Women

Oxidized Phospholipids, Lipoprotein(a), Lipoprotein-Associated Phospholipase A2 Activity, and 10-Year Cardiovascular Outcomes: Prospective Results From the Bruneck Study

Serum Adipocyte Fatty Acid-Binding Protein Levels Were Independently Associated With Carotid Atherosclerosis

A Systematic Review and Meta-Analysis of Statin Therapy in Children With Familial Hypercholesterolemia

Weight-Related Increases in Hypertension, Hypercholesterolemia, and Diabetes Risk in Normal Weight Male and Female Runners

Relationship Between a Systemic Inflammatory Marker, Plaque Inflammation, and Plaque Characteristics Determined by Intravascular Optical Coherence Tomography

Minimal Lipidation of Pre-ß HDL by ABCA1 Results in Reduced Ability to Interact with ABCA1

In Vivo Reverse Cholesterol Transport From Macrophages Lacking ABCA1 Expression Is Impaired

Preferential Sphingosine-1-Phosphate Enrichment and Sphingomyelin Depletion Are Key Features of Small Dense HDL3 Particles: Relevance to Antiapoptotic and Antioxidative Activities

Thrombosis

Effects of Unfractionated Heparin and Glycoprotein IIb/IIIa Antagonists Versus Bivalirdin on Myeloperoxidase Release From Neutrophils

A Novel Class of Antioxidants Inhibit LPS Induction of Tissue Factor by Selective Inhibition of the Activation of ASK1 and MAP Kinases

Letters to the Editor

Does Cuff Location for FMD Matter in Smokers?

Does Cuff Location for FMD Matter in Smokers?

Reconstituted HDL Increases Circulating Endothelial Progenitor Cells in Patients With Type 2 Diabetes

Prevalence of Cholesteryl Ester Storage Disease

Spotlight

Interferon- ${gamma}$ Suppresses Cyclooxygenase-2 Promoter Activity by Inhibiting C-Jun and C/EBPß Binding

TNF- ${alpha}$ Suppresses Prolyl-4-Hydroxylase ${alpha}$ 1 Expression via the ASK1–JNK–NonO Pathway

Increased Inhibition of Inward Rectifier K⁺ Channels by Angiotensin II in Small-Diameter Coronary Artery of Isoproterenol-Induced Hypertrophied Model