In the article by Sutliff et al, which appeared in the September 2013 issue of the journal (Arterioscler Thromb Vasc Biol. 2013;33:2154–2161. DOI: 10.1161/ATVBAHA.113.301913), the legend for Figure 4 should have appeared as:
Disrupted elastic lamellae (EL) and increased extracellular matrix in Poldip2+/− aorta. Transmission electron micrographs of transverse aortic sections from wild-type (top) and Poldip2+/− (bottom) mice at increasing magnifications (left to right). The vascular lumen is visible at the top of images A to D. EL appear dark after poststaining with tannic acid in A and B. Breaks in elastic lamellae in Poldip2+/− are marked with arrows in B and D. Morphometric measurements of interlamellar amorphous (C and D) or fibrillar (F) extracellular matrix (ECM) areas in 195 images from 3 wild-type and 3 Poldip2+/− mice are expressed as average±SEM in E; *P<0.05. Poldip2 indicates polymerase delta interacting protein 2; and VSMC, vascular smooth muscle cell.
The online version of the article has been corrected and is available at http://atvb.ahajournals.org/content/33/9/2154.full.
- © 2013 American Heart Association, Inc.