Donate Help Contact The AHA Sign In Home
American Heart Association
Arteriosclerosis, Thrombosis, and Vascular Biology
Search: search_blue_button Advanced Search
Correction for Caligiuri et al., Arterioscler Thromb Vasc Biol 25 (4) 655-657.
Arteriosclerosis, Thrombosis, and Vascular Biology. 2005;25:e140

This Article
Right arrow Full Text
Right arrow Full Text (PDF)
Right arrow Submit a response
Right arrow Alert me when this article is cited
Right arrow Alert me when eLetters are posted
Right arrow Alert me if a correction is posted
Services
Right arrow Email this article to a friend
Right arrow Similar articles in this journal
Right arrow Alert me to new issues of the journal
Right arrow Download to citation manager
Right arrowRequest Permissions
Citing Articles
Right arrow Citing Articles via Google Scholar
Google Scholar
Right arrow Search for Related Content
(Arteriosclerosis, Thrombosis, and Vascular Biology. 2005;25:e140.)
© 2005 American Heart Association, Inc.

Correction


An extract of the first 100% of the full text is provided, because this article has no abstract.
 

In the April 2005 issue of Arteriosclerosis, Thrombosis, and Vascular Biology, in the editorial by Caligiuri et al (Arterioscler Thromb Vasc Biol. 2005:25:655–657.), a reference was mistakenly omitted from the first line of the fourth paragraph. The complete statement, with the appropriate citation and reference, is listed below. The authors regret this error.

Previous studies have shown that blocking IL–18 signaling by overexpression of the IL–18 binding protein (the endogenous inhibitor of IL–18) prevents fatty streak formation and slows progression to mature plaque.14a

14aMallat Z, Corbaz A, Scoazec A, Graber P, Alouani S, Esposito B, Humbert Y, Chvatchko Y, Tedgui A. Interleukin–18/interleukin–18 binding protein signaling modulates atherosclerotic lesion development and stability. Circ Res.2001;89:e41–e45.